Fluoxetine potentiation of methylphenidate-induced gene regulation in striatal output pathways: Potential role for 5-HT1B receptor

Vincent van Waes; Sarah Ehrlich; Joel Beverley; Heinz Steiner

doi:10.1016/j.neuropharm.2014.08.024

Journal Articles Neuropharmacology Year : 2015

Fluoxetine potentiation of methylphenidate-induced gene regulation in striatal output pathways: Potential role for 5-HT1B receptor

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Vincent van Waes

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Laboratoires de neurosciences intégratives et cliniques (EA 481)

Sarah Ehrlich

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Joel Beverley

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Heinz Steiner

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Life Sciences [q-bio] Neurons and Cognition [q-bio.NC]

Vincent Van Waes : Connect in order to contact the contributor

https://hal-univ-fcomte.archives-ouvertes.fr/hal-03626365

Submitted on : Thursday, March 31, 2022-2:51:32 PM

Last modification on : Friday, August 5, 2022-9:24:09 AM

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hal-03626365 , version 1 (31-03-2022)

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HAL Id : hal-03626365 , version 1
DOI : 10.1016/j.neuropharm.2014.08.024
PUBMEDCENTRAL : PMC4250300

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Vincent van Waes, Sarah Ehrlich, Joel Beverley, Heinz Steiner. Fluoxetine potentiation of methylphenidate-induced gene regulation in striatal output pathways: Potential role for 5-HT1B receptor. Neuropharmacology, 2015, 89, pp.77-86. ⟨10.1016/j.neuropharm.2014.08.024⟩. ⟨hal-03626365⟩

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Fluoxetine potentiation of methylphenidate-induced gene regulation in striatal output pathways: Potential role for 5-HT1B receptor

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